Dr Sara Farrah Heuss

Centre for Tumour Biology
Postdoctoral Research Assistant
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C-Met is a receptor tyrosine kinase, overexpressed in pancreatic cancer. Studies published by our group have shown that c-Met triggers the activation of oncogenic signals such as ERK1/2, STAT3 and Rac1 from endosomes.

My work focuses on c-Met signalling on endosomes in pancreatic cancer, and to evaluate how it can be exploited to benefit pancreatic cancer patients.

See other researchers working on:

Cell Signalling Pancreas
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